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+A 63 year old patient who reported a visual acuity (VA) deficit in the right eye (OD) of several months' evolution.
+His personal history included post-traumatic epilepsy of ten years' duration, arterial hypertension and type II diabetes mellitus.
+He had been treated with 1.5 g vigabatrin and 200 mg carbamazepine daily since the trauma.
+VA was 0.3 in the OD and 0.8 in the left eye with correction.
+Anterior biomicroscopy and tonometry were normal.
+Ophthalmoscopy revealed pallor of both papillae and a macular epiretinal membrane in the OD.
+
+Computerised Humphrey 30/2 white-white and blue-yellow perimetry showed severe concentric campimetric reductions in both eyes (Humphrey instruments, model 745, Carl Zeiss Inc).
+
+She had no family history of interest, so hereditary optic neuropathy was ruled out.
+The anamnesis and analytical determinations of mean corpuscular volume, vitamins B1, B6, B12 and folic acid ruled out nutritional aetiology.
+A diagnosis of retinotoxicity due to vigabatrin was established and the patient was referred to the neurology department.
+At one year of follow-up, the loss of campimetry and VA persisted.
+
+